← Leaderboards
Robert Owen Watson
Texas A&M Agrilife Research
$4,590,757
Attributed
$8,420,224
Total exposure
6
Grants
5
Lead (contact PI)
Attributed= this PI's even-split share of every grant they're on (the fair, additive number). Exposure = full size of all those grants.
Funding over time
peak $1.3M · FY2016–25$2M$1.5M$1M$500K$0
'16
'17
'18
'19
'20
'21
'22
'23
'24
'25
Funding mix
By agency
NIH$8,420,224 · 6
By mechanism
R01$7,612,804 · 4
R21$807,420 · 2
Top collaborators
- Kristin Leigh Patrick11 shared
- Pingwei Li7 shared
- Junjie Zhang5 shared
- Andrew Phillip West2 shared
Most similar at Texas A&M Agrilife Research
Same institution · by research overlap
- Pingwei Li$2,620,833
- Junjie Zhang$4,460,630
Others in their field
Top investigators on “Infection”
- Wafaa M. El-Sadr · Columbia University Health Sciences$965,939,185
- Lawrence Corey · Fred Hutchinson Cancer Center$789,608,171
- Judith S. Currier · University Of California Los Angeles$511,038,212
- Margaret Juliana McElrath · Fred Hutchinson Cancer Center$463,711,983
- Richard Marlink · Elizabeth Glaser Pediatric Aids Fdn$457,210,249
- Joseph J. Eron · Univ Of North Carolina Chapel Hill$415,229,497
Research focus
InfectionPathway InteractionsPathogenCellsPathogenesisMacrophageProteinsMolecularInterferon Type IBaseTuberculosisGenesDs-DnaPhosphorylationInnate Immune ResponseMouse ModelMediatingMycobacterium TuberculosisProductionComplexInnovationBacteriaImmune ResponseExperimental Study
Grant awards (22)
Targeting mediators of oxidative stress to limit hyperinflammatory cell death modalities during Mycobacterium tuberculosis infection$769,168
R01 · FY2025 · AI · contact PI
Mitochondria as crucial regulators of innate immune outcomes during Mycobacterium tuberculosis infection$577,628
R01 · FY2025 · AI · contact PI
Targeting mediators of oxidative stress to limit hyperinflammatory cell death modalities during Mycobacterium tuberculosis infection$660,642
R01 · FY2024 · AI · contact PI
Mitochondria as crucial regulators of innate immune outcomes during Mycobacterium tuberculosis infection$579,583
R01 · FY2024 · AI · contact PI
Mitochondria as crucial regulators of innate immune outcomes during Mycobacterium tuberculosis infection$1
R01 · FY2024 · AI · contact PI
Targeting mediators of oxidative stress to limit hyperinflammatory cell death modalities during Mycobacterium tuberculosis infection$1
R01 · FY2024 · AI · contact PI
Mitochondria as crucial regulators of innate immune outcomes during Mycobacterium tuberculosis infection$543,571
R01 · FY2023 · AI · contact PI
Molecular basis of viral DNA sensing through the cGAS-STING pathway$360,879
R01 · FY2023 · AI
Mitochondria as crucial regulators of innate immune outcomes during Mycobacterium tuberculosis infection$525,006
R01 · FY2022 · AI · contact PI
Molecular basis of viral DNA sensing through the cGAS-STING pathway$361,373
R01 · FY2022 · AI
Mitochondria as crucial regulators of innate immune outcomes during Mycobacterium tuberculosis infection$663,099
R01 · FY2021 · AI · contact PI
Molecular basis of viral DNA sensing through the cGAS-STING pathway$361,843
R01 · FY2021 · AI
Molecular basis of viral DNA sensing through the cGAS-STING pathway$362,292
R01 · FY2020 · AI
TRIM proteins polarize DNA sensing outcomes during the innate immune response to Mycobacterium tuberculosis$371,250
R01 · FY2019 · AI · contact PI
Molecular basis of viral DNA sensing through the cGAS-STING pathway$362,718
R01 · FY2019 · AI
The secreted effector SseC is a key regulator of retrograde transport during Salmonella enterica serovar Typhimurium infection$180,846
R21 · FY2019 · AI · contact PI
TRIM proteins polarize DNA sensing outcomes during the innate immune response to Mycobacterium tuberculosis$371,250
R01 · FY2018 · AI · contact PI
The secreted effector SseC is a key regulator of retrograde transport during Salmonella enterica serovar Typhimurium infection$218,199
R21 · FY2018 · AI · contact PI
TRIM proteins polarize DNA sensing outcomes during the innate immune response to Mycobacterium tuberculosis$371,250
R01 · FY2017 · AI · contact PI
The splicing factor SF3b1 is a master regulator of the innate immune response to Mycobacterium tuberculosis$185,625
R21 · FY2017 · AI · contact PI
TRIM proteins polarize DNA sensing outcomes during the innate immune response to Mycobacterium tuberculosis$371,250
R01 · FY2016 · AI · contact PI
The splicing factor SF3b1 is a master regulator of the innate immune response to Mycobacterium tuberculosis$222,750
R21 · FY2016 · AI · contact PI