← Leaderboards
Kristin Leigh Patrick
Texas A&M University
$4,916,534
Attributed
$7,539,931
Total exposure
4
Grants
1
Lead (contact PI)
Attributed= this PI's even-split share of every grant they're on (the fair, additive number). Exposure = full size of all those grants.
Funding over time
peak $1.8M · FY2018–25$2M$1.5M$1M$500K$0
'18
'19
'20
'21
'22
'23
'24
'25
Funding mix
By agency
NIH$7,539,931 · 4
By mechanism
R01$4,318,699 · 2
R35$2,822,187 · 1
R21$399,045 · 1
Top collaborators
- Robert Owen Watson11 shared
- Pingwei Li2 shared
- Andrew Phillip West2 shared
Most similar at Texas A&M University
Same institution · by research overlap
- Ping He$3,470,539
- Andrew Phillip West$2,424,011
Others in their field
Other Emerging Leaders on “Macrophage”
- Ethan Dmitrovsky · Leidos Biomedical Research, Inc.$20,250,477
- Frederic Geissmann · Sloan-Kettering Inst Can Research$12,957,901
- Adam David Hoppe · South Dakota State University$9,046,059
- Niccolo Terrando · Duke University$6,839,733
- Markus Bosmann · Boston University Medical Campus$6,657,387
- Benson Edagwa · University Of Nebraska Medical Center$6,144,379
Research focus
MacrophageExperimental StudyMolecularPathogenPhosphorylationMediatingImmuneInfectionCellsPlayPathway InteractionsPathogenesisAutomobile DrivingProteinsMutationLinkParkinson DiseaseInflammatoryInflammationIn VivoMouse ModelStressComplexInsight
Grant awards (18)
Targeting mediators of oxidative stress to limit hyperinflammatory cell death modalities during Mycobacterium tuberculosis infection$769,168
R01 · FY2025 · AI
Mitochondria as crucial regulators of innate immune outcomes during Mycobacterium tuberculosis infection$577,628
R01 · FY2025 · AI
Exploring the role of RNA binding proteins in post-transcriptional regulation of macrophage activation$480,631
R35 · FY2025 · GM · contact PI
Targeting mediators of oxidative stress to limit hyperinflammatory cell death modalities during Mycobacterium tuberculosis infection$660,642
R01 · FY2024 · AI
Mitochondria as crucial regulators of innate immune outcomes during Mycobacterium tuberculosis infection$579,583
R01 · FY2024 · AI
Exploring the role of RNA binding proteins in post-transcriptional regulation of macrophage activation$480,631
R35 · FY2024 · GM · contact PI
Mitochondria as crucial regulators of innate immune outcomes during Mycobacterium tuberculosis infection$1
R01 · FY2024 · AI
Targeting mediators of oxidative stress to limit hyperinflammatory cell death modalities during Mycobacterium tuberculosis infection$1
R01 · FY2024 · AI
Mitochondria as crucial regulators of innate immune outcomes during Mycobacterium tuberculosis infection$543,571
R01 · FY2023 · AI
Pre-mRNA splicing regulation is critical for controlling macrophage activation$369,916
R35 · FY2023 · GM · contact PI
Mitochondria as crucial regulators of innate immune outcomes during Mycobacterium tuberculosis infection$525,006
R01 · FY2022 · AI
Pre-mRNA splicing regulation is critical for controlling macrophage activation$370,337
R35 · FY2022 · GM · contact PI
Mitochondria as crucial regulators of innate immune outcomes during Mycobacterium tuberculosis infection$663,099
R01 · FY2021 · AI
Pre-mRNA splicing regulation is critical for controlling macrophage activation$370,738
R35 · FY2021 · GM · contact PI
Pre-mRNA splicing regulation is critical for controlling macrophage activation$374,934
R35 · FY2020 · GM · contact PI
Pre-mRNA splicing regulation is critical for controlling macrophage activation$375,000
R35 · FY2019 · GM · contact PI
The secreted effector SseC is a key regulator of retrograde transport during Salmonella enterica serovar Typhimurium infection$180,846
R21 · FY2019 · AI
The secreted effector SseC is a key regulator of retrograde transport during Salmonella enterica serovar Typhimurium infection$218,199
R21 · FY2018 · AI