DTI IN WILLIAMS SYNDROME

$12,030P41FY2009RRNIH

Stanford University, Stanford CA

Investigators

Linked publications, trials & patents

Paper 36935627 Paper 35864992 Paper 32783339 Paper 32329076 Paper 31722599 Paper 31277060 Paper 30637847 Paper 28792653 Paper 28722314 Paper 28450978 Paper 28137627 Paper 27726251 Paper 27678106 Paper 27282860 Paper 27116907 Paper 26970093 Paper 26923108 Paper 26786745 Paper 26778549 Paper 26774615 Paper 26677408 Paper 26315797 Paper 26026643 Paper 25983363 Paper 25982242 Paper 25970596 Paper 25923677 Paper 25426935 Paper 25400102 Paper 25338633 Paper 25278065 Paper 25193002 Paper 25155435 Paper 25107618 Paper 24907854 Paper 24715414 Paper 24549830 Paper 24512800 Paper 24493404 Paper 24419985 Paper 24324530 Paper 24321559 Paper 24248734 Paper 24227703 Paper 24224182 Paper 24166577 Paper 24089369 Paper 23821305 Paper 23796090 Paper 23661904 Paper 23649942 Paper 23622097 Paper 23617511 Paper 23559437 Paper 23553967 Patent 9170312 Patent 8638096 Patent 8587311 Patent 8538115 Patent 8508225 Patent 8473536 Patent 8432166 Patent 7966053 Patent 7952353 Patent 7928729 Patent 7924002 Patent 7821263 Patent 7795868 Patent 7777486 Patent 7709266 Patent 7683618 Patent 7602183 Patent 7592807 Patent 7592806 Patent 7535222 Patent 7439739 Patent 7368910 Patent 7349729 Patent 7348776 Patent 7298144 Patent 7202662 Patent 7176683 Patent 7132828 Patent 7096056 Patent 6976953 Patent 6975895 Patent 6969991 Patent 6856134 Patent 6853191 Patent 6812700 Patent 6810110 Patent 6806709 Patent 6784664 Patent 6529002 Patent 6463315 Patent 6445181 Patent 6353752 Patent 6324240 Patent 6268728 Patent 6144873

Abstract

This subproject is one of many research subprojects utilizing the resources provided by a Center grant funded by NIH/NCRR. The subproject and investigator (PI) may have received primary funding from another NIH source, and thus could be represented in other CRISP entries. The institution listed is for the Center, which is not necessarily the institution for the investigator. Cognitive hallmarks of Williams syndrome (WS) include severe visuospatial deficits, and relative strengths in face and object processing (Meyer-Lindenberg et al., 2006). Functional neuroimaging data has indicated anomalous functional connectivity of the dorsal stream in WS. Specific Aims: To investigate whether WS has aberrant connectivity of dorsal stream white matter tracts which may link between genetic and visuospatial abnormalities in WS. Methods: We used diffusion tensor imaging to examine white matter integrity in the dorsal and ventral streams among individuals with Williams syndrome (WS) compared to two control groups (typically developing and developmentally delayed) and using three separate analysis methods (whole brain, region of interest, and fiber tractography).

View original record on NIH RePORTER →