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Rui-Ping Xiao
Aging
$0
Attributed
$0
Total exposure
0
Grants
0
Lead (contact PI)
Attributed= this PI's even-split share of every grant they're on (the fair, additive number). Exposure = full size of all those grants.
Funding mix
By agency
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By mechanism
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Top collaborators
No co-investigators on record.
Others in their field
Top investigators on “Mediating”
- Bambra Strokes · Ppd Development Lp$526,656,217
- Lawrence Corey · Fred Hutchinson Cancer Center$493,822,581
- Barton F Haynes · Duke University$295,802,493
- Glenda E Gray · Wits Health Consortium (Pty), Ltd$286,700,869
- Margaret Juliana McElrath · Fred Hutchinson Cancer Center$176,977,598
- Dan H. Barouch · Fred Hutchinson Cancer Center$122,734,499
Research focus
MediatingPlayResponseSignal TransductionHeartProteinsCardiacPhosphorylationMuscle CellsAdultPathway InteractionsHeart FailureGene TransferApoptosisMyocytes, CardiacReceptorCouplingGtp-Binding ProteinsCell DeathPhysiologicalAdrenergic ReceptorOxidative StressCardiac MyocytesMyocardial Infarction
Grant awards (28)
Dimerization of Beta1-Adrenergic Receptor with RAGE Induces Heart Cell Death$225,888
ZIA · FY2009 · AG · contact PI
Agonist Stereochemistry Determines Beta2-Adrenergic G Protein Coupling$225,888
ZIA · FY2009 · AG · contact PI
RGS2 Constitutes A Negative Regulator of Beta2-Adrenergic Gi Signaling$225,888
ZIA · FY2009 · AG · contact PI
Suppression of Beta-arrestin1 Phosphorylation and Function by Beta-arrestin2$203,300
ZIA · FY2009 · AG · contact PI
Cardioprotection by CaMKII-dB Is Mediated by Inducible HSP70$192,005
ZIA · FY2009 · AG · contact PI
B-Arrestin2 Is Required for BAR Resensitization But Not its Desensitization$379,424
Z01 · FY2008 · AG · contact PI
Mitochondrial Protein HSG (Mfn-2) Is A Primary Mediator of Oxidative Apoptosis$379,424
Z01 · FY2008 · AG · contact PI
RGS2 Constitutes A Negative Regulator of Beta2-Adrenergic Gi Signaling$293,193
Z01 · FY2008 · AG · contact PI
CaMKII-dB and CaMKII-dC Oppositely Regulate Cardiomyocyte viability$284,568
Z01 · FY2008 · AG · contact PI
Agonist Stereochemistry Determines b2-Adrenergic G Protein Coupling Preference$198,337
Z01 · FY2008 · AG · contact PI
CaMKII-dB and CaMKII-dC Oppositely Regulate Cardiomyocyte viability$654,709
Z01 · FY2007 · AG · contact PI
Mitochondrial Protein HSG Is A Primary Mediator of Oxidative Apoptotic Death$561,179
Z01 · FY2007 · AG · contact PI
B-Arrestin2 Is Required for BAR Resensitization But Not its Desensitization$467,649
Z01 · FY2007 · AG · contact PI
CaMKII-aB Protects the Heart Against Oxidative-Stress In$0
Z01 · FY2006 · AG · contact PI
Heterodimer of b1-AR and b2-AR Optimizes b-AR Modulation$0
Z01 · FY2006 · AG · contact PI
B-Arrestin2 Is Required for BAR Resensitization But Not$0
Z01 · FY2006 · AG · contact PI
Mitochondrial Protein HSG Is a Major Determinant of Oxid$0
Z01 · FY2006 · AG · contact PI
Dysregulation of HSG Triggers Cardiomyocyte Apoptosis$0
Z01 · FY2005 · AG
Heterodimer of b1-AR and b2-AR Optimizes b-AR Modulation$0
Z01 · FY2005 · AG
CaMKII dB and dC have Opposing roles in regulating fate$0
Z01 · FY2005 · AG
PI3K Offsets b1-Adrenoceptor/PKA-Mediated Positive Inotr$0
Z01 · FY2004 · AG
Intracellular Acidosis-Activated p38 MAPK &Hypoxia$0
Z01 · FY2004 · AG
Dysregulation of HSG Triggers Proliferative Disorders$0
Z01 · FY2004 · AG
Identification and Characterization of A Novel Gene, HSG$0
Z01 · FY2003 · AG
Beta1-adrenergic Apoptotic Signal Delivered By CaMKII$0
Z01 · FY2003 · AG
Cardiac Excitation-Contraction Coupling by p38 MAPK$0
Z01 · FY2003 · AG
Pi3k Compartmentalizes Gs Signaling During B2-adrenergic$0
Z01 · FY2003 · AG
Pi3k Gs Signal Control During B2-adrenergic stimulation$0
Z01 · FY2002 · AG