← Leaderboards
Kenneth Humphries
University Of California San Diego
$5,952,275
Attributed
$5,952,275
Total exposure
8
Grants
7
Lead (contact PI)
Attributed= this PI's even-split share of every grant they're on (the fair, additive number). Exposure = full size of all those grants. They are the sole PI on all grants (the two match).
Funding over time
peak $973K · FY2010–25$1M$750K$500K$250K$0
'10
'11
'12
'13
'14
'15
'16
'17
'18
'19
'20
'21
'22
'23
'24
'25
Funding mix
By agency
NIH$5,952,275 · 8
By mechanism
R01$3,998,985 · 3
P20$1,408,310 · 3
R21$480,700 · 1
F32$64,280 · 1
Top collaborators
No co-investigators on record.
Most similar at University Of California San Diego
Same institution · by research overlap
- Wolfgang H Dillmann$17,750,136
- Teresa Dianne Langford$10,314,213
- W D Parker$1,138,133
- Deborah Hye Spector$13,137,426
- Robert Keith Naviaux$394,130
Others in their field
Top investigators on “Mitochondria”
- Bruce M Spiegelman · Dana-Farber Cancer Inst$30,163,696
- Shi Du Yan · Columbia University Health Sciences$29,685,201
- Peipei Ping · University Of California Los Angeles$27,344,561
- Mary McGrae McDermott · Northwestern University$25,752,019
- Vamsi Krishna Mootha · Massachusetts General Hospital$23,004,991
- Kenneth D. Tew · Fox Chase Cancer Center$22,000,362
Research focus
MitochondriaPreventCardiacProductionEnzymesMetabolicOxidationDiabetes MellitusCardiac MyocytesDiabetic CardiomyopathyFlexibilityInsulin-Dependent Diabetes MellitusMediatingMitochondrial DysfunctionBioenergeticsHyperglycemiaMetabolismHeart DiseasesFunctional DisorderCause Of DeathRegulationAffectFree RadicalsHeart
Grant awards (20)
A novel role for oxidized lipid mediators as effectors of muscle atrophy and weakness in aging$535,985
R01 · FY2025 · AG · contact PI
Increasing glycolysis in the diabetic heart is cardioprotective and improves glucose tolerance$437,000
R01 · FY2025 · HL · contact PI
Increasing glycolysis in the diabetic heart is cardioprotective and improves glucose tolerance$437,000
R01 · FY2024 · HL · contact PI
Increasing glycolysis in the diabetic heart is cardioprotective and improves glucose tolerance$437,000
R01 · FY2023 · HL · contact PI
Investigating the role of SIRT3 in metabolic flexibility and proteostasis in the aging heart$218,500
R21 · FY2023 · AG · contact PI
Increasing glycolysis in the diabetic heart is cardioprotective and improves glucose tolerance$437,000
R01 · FY2022 · HL · contact PI
Investigating the role of SIRT3 in metabolic flexibility and proteostasis in the aging heart$262,200
R21 · FY2022 · AG · contact PI
PKA Signaling and Metabolic Inflexibility in the Diabetic Heart$428,750
R01 · FY2019 · HL · contact PI
PKA Signaling and Metabolic Inflexibility in the Diabetic Heart$428,750
R01 · FY2018 · HL · contact PI
PKA Signaling and Metabolic Inflexibility in the Diabetic Heart$428,750
R01 · FY2017 · HL · contact PI
PKA Signaling and Metabolic Inflexibility in the Diabetic Heart$428,750
R01 · FY2016 · HL · contact PI
Project 1 Mechanisms of Mitochondrial Dysfunction in Diabetic Cardiomyopathy$218,410
P20 · FY2016 · GM · contact PI
Project 1 Mechanisms of Mitochondrial Dysfunction in Diabetic Cardiomyopathy$229,374
P20 · FY2015 · GM · contact PI
Project 1 Mechanisms of Mitochondrial Dysfunction in Diabetic Cardiomyopathy$234,215
P20 · FY2014 · GM · contact PI
Project 1$238,619
P20 · FY2013 · GM · contact PI
Project 1$254,250
P20 · FY2012 · GM · contact PI
MITOCHONDRIAL DYSFUNCTION IN DIABETIC CARDIOMYOPATHY$160,376
P20 · FY2011 · RR · contact PI
THE ROLE OF COMPLEX 1 IN MITOCHONDRIAL DYSFUNCTION & FREE RADICAL PROD IN TYPE 1$73,066
P20 · FY2010 · RR · contact PI
Role of DAKAPs in Mitochondrial Function$25,960
F32 · FY2003 · GM
Role of DAKAPs in Mitochondrial Function$38,320
F32 · FY2002 · GM