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Colin Robert Reily
University Of Alabama At Birmingham
$2,556,945
Attributed
$2,556,945
Total exposure
3
Grants
3
Lead (contact PI)
Attributed= this PI's even-split share of every grant they're on (the fair, additive number). Exposure = full size of all those grants. They are the sole PI on all grants (the two match).
Funding over time
peak $490.6K · FY2015–25$500K$375K$250K$125K$0
'15
'16
'17
'18
'19
'20
'21
'22
'23
'24
'25
Funding mix
By agency
NIH$2,556,945 · 3
By mechanism
R01$1,416,470 · 1
K01$917,725 · 1
R03$222,750 · 1
Top collaborators
No co-investigators on record.
Most similar at University Of Alabama At Birmingham
Same institution · by research overlap
- Jan Novak$14,197,925
- Matthew B Renfrow$7,936,462
- Jiri F Mestecky$6,392,348
- Suzanne M. Michalek$6,713,138
- Kohtaro Fujihashi$5,618,080
Others in their field
Top investigators on “Enzymes”
- Ethan Dmitrovsky · Leidos Biomedical Research, Inc.$143,577,666
- Randall J Bateman · Washington University$83,824,814
- Stuart L Schreiber · Harvard University (Medical School)$49,424,554
- Jay D Keasling · University Of California-Berkeley$47,558,861
- David Heimbrook · Leidos Biomedical Research, Inc.$47,513,682
- Susan Abushakra · Alzheon, Inc.$47,265,244
Research focus
EnzymesAntigen-Antibody ComplexSignal TransductionCytokineGlycosylationIga1ImmuneProductionB-LymphocytesAutoantigensCellsDepositionImmunoglobulin AInjuryGalactoseDisease ProgressionInterleukin-6Pathway InteractionsEnd Stage Renal FailureAutoantibodiesKidneyHematuriaAffectGlycosyltransferase
Grant awards (11)
Distinct Glycophenotypes with Abnormal Signaling Define a Subpopulation of B cells Responsible for Production of Galactose-Deficient IgA1, the Main Autoantigen in IgA Nephropathy$448,004
R01 · FY2025 · DK · contact PI
Distinct Glycophenotypes with Abnormal Signaling Define a Subpopulation of B cells Responsible for Production of Galactose-Deficient IgA1, the Main Autoantigen in IgA Nephropathy$477,871
R01 · FY2024 · DK · contact PI
Distinct Glycophenotypes with Abnormal Signaling Define a Subpopulation of B cells Responsible for Production of Galactose-Deficient IgA1, the Main Autoantigen in IgA Nephropathy$490,595
R01 · FY2023 · DK · contact PI
Abnormal STAT3 Signaling and Aberrant O-Glycosylation of IgA1 in IgA Nephropathy$152,704
K01 · FY2021 · DK · contact PI
Abnormal cytokine response and autoantigen production in IgA-producing subpopulations in IgA nephropathy$111,375
R03 · FY2020 · DK · contact PI
Abnormal STAT3 signaling and aberrant O-glycosylation of IgA1 in IgA nephropathy$152,704
K01 · FY2019 · DK · contact PI
Abnormal cytokine response and autoantigen production in IgA-producing subpopulations in IgA nephropathy$111,375
R03 · FY2019 · DK · contact PI
Abnormal STAT3 signaling and aberrant O-glycosylation of IgA1 in IgA nephropathy$152,704
K01 · FY2018 · DK · contact PI
Abnormal STAT3 signaling and aberrant O-glycosylation of IgA1 in IgA nephropathy$152,704
K01 · FY2017 · DK · contact PI
Abnormal STAT3 signaling and aberrant O-glycosylation of IgA1 in IgA nephropathy$152,704
K01 · FY2016 · DK · contact PI
Abnormal STAT3 signaling and aberrant O-glycosylation of IgA1 in IgA nephropathy$154,205
K01 · FY2015 · DK · contact PI