← Leaderboards
Kayla Branyan-Schroer
University Of California, San Francisco
$209,537
Attributed
$209,537
Total exposure
1
Grants
1
Lead (contact PI)
Attributed= this PI's even-split share of every grant they're on (the fair, additive number). Exposure = full size of all those grants. They are the sole PI on all grants (the two match).
Funding over time
peak $68.6K · FY2019–22$100K$75K$50K$25K$0
'19
'20
'21
'22
Funding mix
By agency
NIH$209,537 · 1
By mechanism
F32$209,537 · 1
Top collaborators
No co-investigators on record.
Most similar at University Of California, San Francisco
Same institution · by research overlap
- Hua Su$11,193,749
- Helen Kim$11,471,453
- Edward C Hsiao$11,767,796
- Rong Wang$8,759,152
- Katja Brueckner$2,284,846
Others in their field
Other Emerging Leaders on “Acvrl1 Gene”
- Marie E Faughnan · University Of California, San Francisco$1,862,091
- Stryder Medoah Meadows · Tulane University Of Louisiana$1,819,500
- Kristopher Kahle · Massachusetts General Hospital$1,294,807
- Laura E Kelley · Aikili Biosystems Inc.$200,000
- Jouha Min · Aikili Biosystems Inc.$200,000
- Jennifer Marie Spangle · Dana-Farber Cancer Inst$194,571
Research focus
Acvrl1 GeneAffectAlternative TreatmentAnimal BehaviorAnimal ModelAnimalsAreaArteriesArteriovenous MalformationBaseBlood CapillariesBlood FlowBlood Vessel DevelopmentBlood VesselsBrainCadherin 5CaliberCell CountCell ProliferationCharacteristicsClinically RelevantCurative TreatmentsDimensionsActivin A
Grant awards (4)
Elucidating mechanisms of hereditary hemorrhagic telangiectasia 2 gene-mediated arteriovenous malformation development via eNOS signaling using in vivo two-photon imaging in a preclinical mouse model$14,439
F32 · FY2022 · HL · contact PI
Elucidating mechanisms of hereditary hemorrhagic telangiectasia 2 gene-mediated arteriovenous malformation development via eNOS signaling using in vivo two-photon imaging in a preclinical mouse model$68,562
F32 · FY2021 · HL · contact PI
Elucidating mechanisms of hereditary hemorrhagic telangiectasia 2 gene-mediated arteriovenous malformation development via eNOS signaling using in vivo two-photon imaging in a preclinical mouse model$65,310
F32 · FY2020 · HL · contact PI
Elucidating mechanisms of hereditary hemorrhagic telangiectasia 2 gene-mediated arteriovenous malformation development via eNOS signaling using in vivo two-photon imaging in a preclinical mouse model$61,226
F32 · FY2019 · HL · contact PI