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Wei Chen

University Of Alabama At Birmingham

$4,097,564
Attributed
$4,097,564
Total exposure
3
Grants
3
Lead (contact PI)

Attributed= this PI's even-split share of every grant they're on (the fair, additive number). Exposure = full size of all those grants. They are the sole PI on all grants (the two match).

Funding over time

peak $763.8K · FY201723
$1M$750K$500K$250K$0
'17
'18
'19
'20
'21
'22
'23

Funding mix

By agency

NIH$4,097,564 · 3

By mechanism

R01$3,726,314 · 2
R56$371,250 · 1

Top collaborators

No co-investigators on record.

Most similar at University Of Alabama At Birmingham

Same institution · by research overlap

Others in their field

Other Emerging Leaders on “Aging

Research focus

AgingMouse ModelDesignSignal TransductionOverexpressionInsightGain Of FunctionGenesPathologicSignal PathwayPhysiologicalPhenotypeBaseLoss Of FunctionInflammationNovel Therapeutic InterventionBeta CateninAffectCartilage RegenerationAging PopulationCartilage RepairEarly OnsetCandidate Disease GeneArticular Cartilage

Grant awards (11)

Cbf mediates articular cartilage regeneration and repair in aging$416,909
R01 · FY2023 · AG · contact PI
Cbf mediates articular cartilage regeneration and repair in aging$416,909
R01 · FY2022 · AG · contact PI
Cbf mediates articular cartilage regeneration and repair in aging$416,909
R01 · FY2021 · AG · contact PI
Characterizing the mechanism by which endogenous negative regulators of osteoclasts control bone homeostasis under physiological and pathological conditions in mouse models$324,368
R01 · FY2021 · AR · contact PI
Cbfβ mediates articular cartilage regeneration and repair in aging$407,309
R01 · FY2020 · AG · contact PI
Characterizing the mechanism by which endogenous negative regulators of osteoclasts control bone homeostasis under physiological and pathological conditions in mouse models$326,700
R01 · FY2020 · AR · contact PI
Cbfβ mediates articular cartilage regeneration and repair in aging$437,110
R01 · FY2019 · AG · contact PI
Characterizing the mechanism by which endogenous negative regulators of osteoclasts control bone homeostasis under physiological and pathological conditions in mouse models$326,700
R01 · FY2019 · AR · contact PI
Characterizing the mechanism by which endogenous negative regulators of osteoclasts control bone homeostasis under physiological and pathological conditions in mouse models$326,700
R01 · FY2018 · AR · contact PI
Cbfβ mediates articular cartilage regeneration and repair in aging$371,250
R56 · FY2017 · AG · contact PI
Characterizing the mechanism by which endogenous negative regulators of osteoclasts control bone homeostasis under physiological and pathological conditions in mouse models$326,700
R01 · FY2017 · AR · contact PI