← Leaderboards
Shyam Sunder Bansal
University Of Alabama At Birmingham
$5,886,236
Attributed
$6,256,792
Total exposure
5
Grants
4
Lead (contact PI)
Attributed= this PI's even-split share of every grant they're on (the fair, additive number). Exposure = full size of all those grants.
Funding over time
peak $1.9M · FY2016–25$2M$1.5M$1M$500K$0
'16
'17
'18
'19
'20
'21
'22
'23
'24
'25
Funding mix
By agency
NIH$6,256,792 · 5
By mechanism
R01$5,275,588 · 3
R00$738,086 · 1
K99$243,118 · 1
Top collaborators
- Isabelle Deschenes1 shared
Most similar at University Of Alabama At Birmingham
Same institution · by research overlap
- Troy D Randall$33,910,350
- Candece L Gladson$9,713,401
- Carlos J Orihuela$11,918,098
- Anoma Nellore$507,411
- Albert F Lobuglio$1,259,317
Others in their field
Top investigators on “Cardiovascular System”
- Sonia M Thomas · Research Triangle Institute$701,865,642
- Tracy L Nolen · Research Triangle Institute$474,487,152
- Chanza Baytop · Westat, Inc.$271,217,917
- Judith S. Currier · University Of California Los Angeles$139,907,800
- Joseph J. Eron · Univ Of North Carolina Chapel Hill$137,892,773
- Judith S Hochman · New York University School Of Medicine$110,228,771
Research focus
Cardiovascular SystemCd4 Positive T LymphocytesScientistCardiacMyocardial DysfunctionHeart FailureMyocardial InfarctionMyocardial IschemiaImmuneHypertrophyHeartCongestive Heart FailureMediatingInflammationLeft Ventricular RemodelingInflammatoryHealingFibrosisCell TypeCellsEchocardiographyImmune ResponseImmunoregulationDisease Progression
Grant awards (16)
Impact of Adaptive Immune System on Cardiac Electrophysiology$741,112
R01 · FY2025 · HL
Novel Inhibitors for Temporal Modulation of T-Lymphocytes during Chronic Heart Failure$609,220
R01 · FY2025 · HL · contact PI
TNFR1 Expressing Exosomes are Critical Mediators of Pathological Immune Activation in the Spleen post-Myocardial Infarction$550,936
R01 · FY2025 · HL · contact PI
Novel Inhibitors for Temporal Modulation of T-Lymphocytes during Chronic Heart Failure$587,161
R01 · FY2024 · HL · contact PI
TNFR1 Expressing Exosomes are Critical Mediators of Pathological Immune Activation in the Spleen post-Myocardial Infarction$559,506
R01 · FY2024 · HL · contact PI
TNFR1 Expressing Exosomes are Critical Mediators of Pathological Immune Activation in the Spleen post-Myocardial Infarction$513,864
R01 · FY2023 · HL · contact PI
Novel Inhibitors for Temporal Modulation of T-Lymphocytes during Chronic Heart Failure$483,883
R01 · FY2023 · HL · contact PI
Novel Inhibitors for Temporal Modulation of T-Lymphocytes during Chronic Heart Failure$89,652
R01 · FY2023 · HL · contact PI
TNFR1 Expressing Exosomes are Critical Mediators of Pathological Immune Activation in the Spleen post-Myocardial Infarction$29,142
R01 · FY2023 · HL · contact PI
TNFR1 Expressing Exosomes are Critical Mediators of Pathological Immune Activation in the Spleen post-Myocardial Infarction$552,356
R01 · FY2022 · HL · contact PI
TNFR1 Expressing Exosomes are Critical Mediators of Pathological Immune Activation in the Spleen post-Myocardial Infarction$558,756
R01 · FY2021 · HL · contact PI
Pathological TNFR1 Expressing CD4+ T-cells are Critical for HF progression$242,713
R00 · FY2020 · HL · contact PI
Pathological TNFR1 Expressing CD4+ T-cells are Critical for HF progression$246,375
R00 · FY2019 · HL · contact PI
Pathological TNFR1 Expressing CD4+ T-cells are Critical for HF progression$248,998
R00 · FY2018 · HL · contact PI
Pathological TNFR1 expressing CD4+ T-cells are critical for HF progression$121,559
K99 · FY2017 · HL · contact PI
Pathological TNFR1 expressing CD4+ T-cells are critical for HF progression$121,559
K99 · FY2016 · HL · contact PI