← Leaderboards
Younghoon Kee
University Of South Florida
$1,640,643
Attributed
$1,640,643
Total exposure
2
Grants
2
Lead (contact PI)
Attributed= this PI's even-split share of every grant they're on (the fair, additive number). Exposure = full size of all those grants. They are the sole PI on all grants (the two match).
Funding over time
peak $439.6K · FY2015–20$500K$375K$250K$125K$0
'15
'16
'17
'18
'19
'20
Funding mix
By agency
NIH$1,640,643 · 2
By mechanism
R01$1,201,033 · 1
R15$439,610 · 1
Top collaborators
No co-investigators on record.
Most similar at University Of South Florida
Same institution · by research overlap
- Xiaohong Mary Zhang$1,801,948
- Sandra D. Westerheide$834,336
Others in their field
Top investigators on “Regulation”
- Kiyoshi Suyehiro · Iodp Management International, Inc$166,539,344
- David Heimbrook · Leidos Biomedical Research, Inc.$152,164,826
- Ralph Parchment · Leidos Biomedical Research, Inc.$141,315,269
- Nevan J Krogan · University Of California, San Francisco$101,337,442
- Asimah Rafi · Technical Resources Inc$95,881,052
- Richard C Boucher · University Of North Carolina Chapel Hill$83,460,414
Research focus
RegulationDeubiquitinating EnzymeLeadPathway InteractionsUbiquitin-Protein LigaseMolecularGenotoxic StressInsightMediatingMonoubiquitinationUbiquitinUbiquitinationBiochemicalResponseCellsBaseChromatinComplexMalignant NeoplasmsDna DamageCell NucleusEpigenetic ProcessAttentionCancer Type
Grant awards (5)
The role of UBR5 in the PRC1-mediated transcriptional repression at damaged chromatin$301,685
R01 · FY2020 · GM · contact PI
The role of UBR5 in the PRC1-mediated transcriptional repression at damaged chromatin$303,285
R01 · FY2019 · GM · contact PI
The role of UBR5 in the PRC1-mediated transcriptional repression at damaged chromatin$291,268
R01 · FY2018 · GM · contact PI
The role of UBR5 in the PRC1-mediated transcriptional repression at damaged chromatin$304,795
R01 · FY2017 · GM · contact PI
Function and Regulation of the Fanconi Anemia pathway in DNA repair$439,610
R15 · FY2015 · HL · contact PI