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Katherine Jean Motyl

Mainehealth

$4,988,053
Attributed
$5,821,007
Total exposure
5
Grants
5
Lead (contact PI)

Attributed= this PI's even-split share of every grant they're on (the fair, additive number). Exposure = full size of all those grants.

Funding over time

peak $859.6K · FY201125
$1M$750K$500K$250K$0
'11
'12
'13
'14
'15
'16
'17
'18
'19
'20
'21
'22
'23
'24
'25

Funding mix

By agency

NIH$5,821,007 · 5

By mechanism

R01$3,646,541 · 2
P20$1,405,197 · 1
K01$612,699 · 1
F32$156,570 · 1

Top collaborators

Most similar at Mainehealth

Same institution · by research overlap

Others in their field

Top investigators on “Osteoclasts

Research focus

OsteoclastsOsteogenesisOsteoblastsBone ResorptionBoneBone LossGenesCellsPathway InteractionsMediatingIn VivoBone DensityBone RemodelingReceptorSignal TransductionFracture RiskSympathetic Nervous SystemBindingBone MarrowIn VitroMarrowPhenotypePreventSerum

Grant awards (24)

Molecular and cellular mechanisms of prevention of bone loss by beta blockers$383,236
R01 · FY2025 · AR · contact PI
A novel cell-autonomous role for β-adrenergic receptor signaling in osteoclasts$326,980
R01 · FY2025 · AR · contact PI
Molecular and cellular mechanisms of prevention of bone loss by beta blockers$413,041
R01 · FY2024 · AR · contact PI
A novel cell-autonomous role for β-adrenergic receptor signaling in osteoclasts$357,766
R01 · FY2024 · AR · contact PI
A novel cell-autonomous role for β-adrenergic receptor signaling in osteoclasts$64,939
R01 · FY2024 · AR · contact PI
Molecular and cellular mechanisms of prevention of bone loss by beta blockers$425,816
R01 · FY2023 · AR · contact PI
A novel cell-autonomous role for β-adrenergic receptor signaling in osteoclasts$368,831
R01 · FY2023 · AR · contact PI
A novel cell-autonomous role for β-adrenergic receptor signaling in osteoclasts$64,939
R01 · FY2023 · AR · contact PI
Molecular and cellular mechanisms of prevention of bone loss by beta blockers$443,816
R01 · FY2022 · AR · contact PI
A novel cell-autonomous role for β-adrenergic receptor signaling in osteoclasts$364,782
R01 · FY2022 · AR · contact PI
A novel cell-autonomous role for β-adrenergic receptor signaling in osteoclasts$50,424
R01 · FY2022 · AR · contact PI
A novel cell-autonomous role for β-adrenergic receptor signaling in osteoclasts$381,971
R01 · FY2021 · AR · contact PI
Project 4: Neural and metabolic mechanisms of atypical antipsychotic drug-induced bone loss$303,861
P20 · FY2020 · GM · contact PI
TRPM8 is a novel regulator of bone homeostasis through neural and cell-autonomous mechanisms$118,518
K01 · FY2020 · AR · contact PI
Project 4: Neural and metabolic mechanisms of atypical antipsychotic drug-induced bone loss$361,236
P20 · FY2019 · GM · contact PI
TRPM8 is a novel regulator of bone homeostasis through neural and cell-autonomous mechanisms$120,884
K01 · FY2019 · AR · contact PI
Project 4: Neural and metabolic mechanisms of atypical antipsychotic drug-induced bone loss$359,497
P20 · FY2018 · GM · contact PI
TRPM8 is a novel regulator of bone homeostasis through neural and cell-autonomous mechanisms$122,067
K01 · FY2018 · AR · contact PI
Project 4: Neural and metabolic mechanisms of atypical antipsychotic drug-induced bone loss$380,603
P20 · FY2017 · GM · contact PI
TRPM8 is a novel regulator of bone homeostasis through neural and cell-autonomous mechanisms$124,432
K01 · FY2017 · AR · contact PI
TRPM8 is a novel regulator of bone homeostasis through neural and cell-autonomous mechanisms$126,798
K01 · FY2016 · AR · contact PI
Mechanisms of Bone Loss from Administration of the Second-Generation Antipsychoti$54,617
F32 · FY2013 · AR · contact PI
Mechanisms of Bone Loss from Administration of the Second-Generation Antipsychoti$52,607
F32 · FY2012 · AR · contact PI
Mechanisms of bone loss from administration of the second-generation antipsychoti$49,346
F32 · FY2011 · AR · contact PI