← Leaderboards
Hiroshi Nishimune
University Of Kansas Medical Center
$4,233,093
Attributed
$5,505,568
Total exposure
9
Grants
9
Lead (contact PI)
Attributed= this PI's even-split share of every grant they're on (the fair, additive number). Exposure = full size of all those grants.
Funding over time
peak $1.4M · FY2007–19$2M$1.5M$1M$500K$0
'07
'08
'09
'10
'11
'12
'13
'14
'15
'16
'17
'18
'19
Funding mix
By agency
NIH$5,505,568 · 9
By mechanism
R01$3,545,413 · 2
S10$987,190 · 1
P20$972,965 · 6
Top collaborators
- Stephen D Meriney5 shared
- John A Stanford5 shared
Most similar at University Of Kansas Medical Center
Same institution · by research overlap
- Jianming Qiu$11,839,179
- Ziying Yan$1,506,776
- Edward Brice Stephens$8,642,647
- Kee Jun Kim$1,578,913
Others in their field
Top investigators on “Proteins”
- David Heimbrook · Leidos Biomedical Research, Inc.$354,061,551
- Randall J Bateman · Washington University$187,292,085
- Paul S. Aisen · Cognition Therapeutics, Inc.$175,382,342
- Reisa A. Sperling · Banner Health$145,046,481
- Barton F Haynes · Duke University$136,029,850
- Joseph L Goldstein · University Of Texas Sw Med Ctr/Dallas$102,433,280
Research focus
ProteinsSynapsesCalcium ChannelBasePresynapticMotor NeuronsResearch PersonnelInsightNervous System DisorderMolecularMaintenanceLamininSynaptic TransmissionAgingAge RelatedPublishingBackNerve SupplyExerciseElectrophysiology (Science)EtiologyImpairmentInnovationBrain
Grant awards (17)
Super resolution microscope for the imaging core facility$987,190
S10 · FY2019 · OD · contact PI
Exercise-activated mechanisms reduce degeneration of aged neuromuscular junctions$379,260
R01 · FY2019 · AG · contact PI
Exercise-activated mechanisms reduce degeneration of aged neuromuscular junctions$379,224
R01 · FY2018 · AG · contact PI
Exercise-activated mechanisms reduce degeneration of aged neuromuscular junctions$379,192
R01 · FY2017 · AG · contact PI
Exercise-activated mechanisms reduce degeneration of aged neuromuscular junctions$379,159
R01 · FY2016 · AG · contact PI
Active zone loss cause dying back neuropathy in amyotrophic lateral sclerosis$330,313
R01 · FY2016 · NS · contact PI
Exercise-activated mechanisms reduce degeneration of aged neuromuscular junctions$391,877
R01 · FY2015 · AG · contact PI
Active zone loss cause dying back neuropathy in amyotrophic lateral sclerosis$330,313
R01 · FY2015 · NS · contact PI
Active zone loss cause dying back neuropathy in amyotrophic lateral sclerosis$327,010
R01 · FY2014 · NS · contact PI
Active zone loss cause dying back neuropathy in amyotrophic lateral sclerosis$318,752
R01 · FY2013 · NS · contact PI
Active zone loss cause dying back neuropathy in amyotrophic lateral sclerosis$330,313
R01 · FY2012 · NS · contact PI
KANSAS U COBRE: ORGANIZATION OF THE NERVE TERMINAL BY SYNAPTIC CLEFT COMPONENTS$220,028
P20 · FY2010 · RR · contact PI
KANSAS U COBRE: ORGANIZATION OF THE NERVE TERMINAL BY SYNAPTIC CLEFT COMPONENTS$220,028
P20 · FY2009 · RR · contact PI
KANSAS U COBRE: ORGANIZATION OF THE NERVE TERMINAL BY SYNAPTIC CLEFT COMPONENTS$215,629
P20 · FY2008 · RR · contact PI
INBRE: KUMC: VOLTAGE GATED CALCIUM CHANNELS AS SCAFFOLDING PROTEINS FOR SYNAPSE$67,581
P20 · FY2008 · RR · contact PI
KANSAS U COBRE: ORGANIZATION OF THE NERVE TERMINAL BY SYNAPTIC CLEFT COMPONENTS$195,764
P20 · FY2007 · RR · contact PI
INBRE: KU-L: VOLTAGE GATED CALCIUM CHANNELS AS SCAFFOLDING PROTEINS FOR SYNAPSE$53,935
P20 · FY2007 · RR · contact PI