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Melissa L Fishel

Indiana Univ-Purdue Univ At Indianapolis

$7,092,542
Attributed
$17,208,751
Total exposure
6
Grants
2
Lead (contact PI)

Attributed= this PI's even-split share of every grant they're on (the fair, additive number). Exposure = full size of all those grants.

Funding over time

peak $2.7M · FY200725
$5M$3.8M$2.5M$1.3M$0
'07
'08
'09
'10
'11
'12
'13
'14
'15
'16
'17
'18
'19
'20
'21
'22
'23
'24
'25

Funding mix

By agency

NIH$17,208,751 · 6

By mechanism

R01$8,927,206 · 3
U01$7,948,245 · 2
R21$333,300 · 1

Top collaborators

Most similar at Indiana Univ-Purdue Univ At Indianapolis

Same institution · by research overlap

Others in their field

Top investigators on “Tumor

Research focus

TumorMalignant Neoplasm Of PancreasTumor GrowthPancreatic Ductal AdenocarcinomaSignal TransductionMalignant NeoplasmsIn VivoGrowthMolecular TargetFibroblastsTumor MicroenvironmentCellsPathway InteractionsOxidation-ReductionTranscription FactorResistanceEvaluationCoculture TechniquesCritical PathwaysSurvival Rate3-DimensionalSignal PathwayCancer EtiologyPharmaceutical Preparations

Grant awards (34)

Reprogramming PDAC Stroma by Targeting Coagulation in the Tumor Microenvironment$932,376
U01 · FY2025 · CA
Metabolic flux analysis and PDX models to understand therapeutic vulnerabilities following inhibition of Ref-1 redox signaling in pancreatic cancer$479,470
R01 · FY2025 · CA
Investigation of novel signaling protein in 3D and in vivo PDAC models using second generation Ref-1 inhibitors$433,463
R01 · FY2025 · CA · contact PI
Metabolic flux analysis and PDX models to understand therapeutic vulnerabilities following inhibition of Ref-1 redox signaling in pancreatic cancer$81,710
R01 · FY2025 · CA
Reprogramming PDAC Stroma by Targeting Coagulation in the Tumor Microenvironment$885,794
U01 · FY2024 · CA
Metabolic flux analysis and PDX models to understand therapeutic vulnerabilities following inhibition of Ref-1 redox signaling in pancreatic cancer$443,001
R01 · FY2024 · CA
Investigation of novel signaling protein in 3D and in vivo PDAC models using second generation Ref-1 inhibitors$411,790
R01 · FY2024 · CA · contact PI
Metabolic flux analysis and PDX models to understand therapeutic vulnerabilities following inhibition of Ref-1 redox signaling in pancreatic cancer$75,358
R01 · FY2024 · CA
Metabolic flux analysis and PDX models to understand therapeutic vulnerabilities following inhibition of Ref-1 redox signaling in pancreatic cancer$27,108
R01 · FY2024 · CA
Reprogramming PDAC Stroma by Targeting Coagulation in the Tumor Microenvironment$670,662
U01 · FY2023 · CA
Metabolic flux analysis and PDX models to understand therapeutic vulnerabilities following inhibition of Ref-1 redox signaling in pancreatic cancer$466,315
R01 · FY2023 · CA
Investigation of novel signaling protein in 3D and in vivo PDAC models using second generation Ref-1 inhibitors$424,794
R01 · FY2023 · CA · contact PI
Reprogramming PDAC Stroma by Targeting Coagulation in the Tumor Microenvironment$257,378
U01 · FY2023 · CA
Reprogramming PDAC Stroma by Targeting Coagulation in the Tumor Microenvironment$956,721
U01 · FY2022 · CA
Targeting the Plasminogen Activation System to Limit Pancreatic Cancer Progression and Associated Thrombosis$834,158
U01 · FY2022 · HL
Exploiting the Ref-1 node in pancreatic cancer: tailoring new pancreatic cancer therapy using multi-targeted combinations$483,852
R01 · FY2022 · CA
Investigation of novel signaling protein in 3D and in vivo PDAC models using second generation Ref-1 inhibitors$424,794
R01 · FY2022 · CA · contact PI
Targeting the Plasminogen Activation System to Limit Pancreatic Cancer Progression and Associated Thrombosis$844,766
U01 · FY2021 · HL
Exploiting the Ref-1 node in pancreatic cancer: tailoring new pancreatic cancer therapy using multi-targeted combinations$493,726
R01 · FY2021 · CA
Investigation of novel signaling protein in 3D and in vivo PDAC models using second generation Ref-1 inhibitors$448,088
R01 · FY2021 · CA · contact PI
Targeting the Plasminogen Activation System to Limit Pancreatic Cancer Progression and Associated Thrombosis$855,078
U01 · FY2020 · HL
Exploiting the Ref-1 node in pancreatic cancer: tailoring new pancreatic cancer therapy using multi-targeted combinations$493,726
R01 · FY2020 · CA
Targeting the Plasminogen Activation System to Limit Pancreatic Cancer Progression and Associated Thrombosis$892,860
U01 · FY2019 · HL
Exploiting the Ref-1 node in pancreatic cancer: tailoring new pancreatic cancer therapy using multi-targeted combinations$563,735
R01 · FY2019 · CA
Targeting the Plasminogen Activation System to Limit Pancreatic Cancer Progression and Associated Thrombosis$818,452
U01 · FY2018 · HL
Exploiting the Ref-1 node in pancreatic cancer: tailoring new pancreatic cancer therapy using multi-targeted combinations$493,726
R01 · FY2018 · CA
Exploiting the Ref-1 node in pancreatic cancer: tailoring new pancreatic cancer therapy using multi-targeted combinations$227,554
R01 · FY2018 · CA
Novel Role of Ref-1 in Pancreatic Cancer Etiology and Progression$493,963
R01 · FY2017 · CA
Novel Role of Ref-1 in Pancreatic Cancer Etiology and Progression$493,963
R01 · FY2016 · CA
Novel Role of Ref-1 in Pancreatic Cancer Etiology and Progression$493,963
R01 · FY2015 · CA
Novel Role of Ref-1 in Pancreatic Cancer Etiology and Progression$479,144
R01 · FY2014 · CA
Novel Role of Ref-1 in Pancreatic Cancer Etiology and Progression$493,963
R01 · FY2013 · CA
Chemosensitization of Pancreatic Tumors via Inhibition of a DNA BER Enzyme, Ape1$181,800
R21 · FY2008 · CA · contact PI
Chemosensitization of Pancreatic Tumors via Inhibition of a DNA BER Enzyme, Ape1$151,500
R21 · FY2007 · CA · contact PI