GGrantIndex
← Leaderboards

Xi-Qin Ding

University Of Oklahoma Hlth Sciences Ctr

$6,463,782
Attributed
$6,463,782
Total exposure
10
Grants
10
Lead (contact PI)

Attributed= this PI's even-split share of every grant they're on (the fair, additive number). Exposure = full size of all those grants. They are the sole PI on all grants (the two match).

Funding over time

peak $632.7K · FY200625
$1M$750K$500K$250K$0
'06
'07
'08
'09
'10
'11
'12
'13
'14
'15
'16
'17
'18
'19
'20
'21
'22
'23
'24
'25

Funding mix

By agency

NIH$6,463,782 · 10

By mechanism

R01$4,706,901 · 3
P20$956,018 · 5
R21$800,863 · 2

Top collaborators

No co-investigators on record.

Most similar at University Of Oklahoma Hlth Sciences Ctr

Same institution · by research overlap

Others in their field

Top investigators on “Retina

Research focus

RetinaRetinal ConeMouse ModelGenesCessation Of LifeInheritedMutationPhotoreceptorsResponseAchromatopsiaVision DisordersInsightBlindnessCyclic-Nucleotide Gated Ion ChannelsConeReceptorApoptosisPhototransductionSignal TransductionOpsinProteinsCytotoxicColor VisionsCyclic Gmp

Grant awards (22)

Thyroid Hormone Signaling and Cone Photoreceptor Degeneration$364,637
R01 · FY2025 · EY · contact PI
Thyroid Hormone Signaling and Cone Photoreceptor Degeneration$364,637
R01 · FY2024 · EY · contact PI
Thyroid Hormone Signaling and Cone Photoreceptor Degeneration$370,245
R01 · FY2023 · EY · contact PI
Thyroid Hormone Signaling and Cone Photoreceptor Degeneration$370,279
R01 · FY2022 · EY · contact PI
The Role of Endoplasmic Reticulum Calcium Channels in Cone Degeneration Resulting from CNG Channel Deficiency$362,500
R01 · FY2020 · EY · contact PI
The Role of Endoplasmic Reticulum Calcium Channels in Cone Degeneration Resulting from CNG Channel Deficiency$362,500
R01 · FY2019 · EY · contact PI
The Role of Endoplasmic Reticulum Calcium Channels in Cone Degeneration Resulting from CNG Channel Deficiency$363,238
R01 · FY2018 · EY · contact PI
The Role of Endoplasmic Reticulum Calcium Channels in Cone Degeneration Resulting from CNG Channel Deficiency$382,929
R01 · FY2017 · EY · contact PI
Suppressing thyroid hormone signaling to protect cones in retinal degeneration$181,300
R21 · FY2015 · EY · contact PI
Mechanism of Cone Degeneration Resulting from CNG Channel Deficiency$348,096
R01 · FY2014 · EY · contact PI
Suppressing thyroid hormone signaling to protect cones in retinal degeneration$222,000
R21 · FY2014 · EY · contact PI
Mechanism of Cone Degeneration Resulting from CNG Channel Deficiency$337,440
R01 · FY2013 · EY · contact PI
Mechanism of Cone Degeneration Resulting from CNG Channel Deficiency$355,200
R01 · FY2012 · EY · contact PI
Mechanism of Cone Degeneration Resulting from CNG Channel Deficiency$355,200
R01 · FY2011 · EY · contact PI
Mechanism of Cone Degeneration Resulting from CNG Channel Deficiency$370,000
R01 · FY2010 · EY · contact PI
STRUCTURE AND FUNCTION RELATIONSHIP OF CONE CYCLIC NUCLEOTIDE-GATED CHANNEL$262,724
P20 · FY2010 · RR · contact PI
STRUCTURE AND FUNCTION RELATIONSHIP OF CONE CYCLIC NUCLEOTIDE-GATED CHANNEL$212,052
P20 · FY2009 · RR · contact PI
STRUCTURE AND FUNCTION RELATIONSHIP OF CONE CYCLIC NUCLEOTIDE-GATED CHANNEL$215,027
P20 · FY2008 · RR · contact PI
Using Nanotechnology to Rescue Visual Loss in A Model of Human Blinding Disorder$177,813
R21 · FY2007 · EY · contact PI
BIOCHEMICAL AND FUNCTIONAL CHARACTERISTICS OF THE CONE CNG CHANNEL$121,910
P20 · FY2007 · RR · contact PI
Using Nanotechnology to Rescue Visual Loss in A Model of Human Blinding Disorder$219,750
R21 · FY2006 · EY · contact PI
BIOCHEMICAL AND FUNCTIONAL CHARACTERISTICS OF THE CONE CNG CHANNEL$144,305
P20 · FY2006 · RR · contact PI