PATHOGENESIS OF FAMILIAL PRIMARY PULMONARY HYPERTENSION

$0M01FY2000RRNIH

Vanderbilt University, Nashville TN

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Abstract

Progress in primary pulmonary hypertension (PPH) and familial primary pulmonary hypertension (FPPH) has led to the development of two general testable hypotheses. 1) Vasoactive mediators in PPH/FPPH will elucidate the pathogenesis of the vascular lesions and 2) restriction fragment length polymorphisms may yield insight into the location and type of genetic defect.

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