SIGNALS THAT PATTERN THE SKULL FLOOR

$0P01FY2002HDNIH

University Of Oregon, Eugene OR

Investigators

Linked publications & trials

Paper 36555308 Paper 35982615 Paper 35143593 Paper 34490846 Paper 32196484 Paper 32112658 Paper 31591228 Paper 31394080 Paper 30850632 Paper 30532251 Paper 30427839 Paper 30398151 Paper 30380418 Paper 30290151 Paper 30287385 Paper 30286167 Paper 30226839 Paper 29777677 Paper 29722095 Paper 29610339 Paper 29475973 Paper 29175679 Paper 28973938 Paper 28874452 Paper 28856758 Paper 28834731 Paper 28658311 Paper 28579318 Paper 28304369 Paper 28207737 Paper 28129860 Paper 28129854 Paper 27960075 Paper 27789622 Paper 27565577 Paper 27507212 Paper 27500400 Paper 27458727 Paper 27351618 Paper 27312493 Paper 27265864 Paper 27195754 Paper 27189439 Paper 27159037 Paper 26814702 Paper 26699284 Paper 26567512 Paper 26507229 Paper 26503773 Paper 26500251 Paper 26453491 Paper 26339860 Paper 26296066 Paper 26061541 Paper 25616960 Paper 25611324 Paper 25516613 Paper 25415969 Paper 25296686 Paper 25294950 Paper 25170046 Paper 25132798 Paper 25044745 Paper 24973743 Paper 24626987 Paper 24554559 Paper 24269905 Paper 24045267 Paper 24040125 Paper 23861062 Paper 23826316 Paper 23747543 Paper 23720234 Paper 23714426 Paper 23698351 Paper 23594743 Paper 23544991 Paper 23471915 Paper 23382349 Paper 23297729 Paper 23242824 Paper 23122226 Paper 23091474 Paper 22983029 Paper 22792396 Paper 22782724 Paper 22761277 Paper 22750409 Paper 22676467 Paper 22627283 Paper 22606267 Paper 22492359 Paper 22390748 Paper 22185793 Paper 22052544 Paper 22016187 Paper 22003409 Paper 21937597 Paper 21914475 Paper 21901110

Abstract

The neurocranium develops from separate domains of pre-skeletal cells. The proposed studies will examine the hypothesis that these domains have separate developmental origins, and that their patterns of morphogenesis are specified by separate conserved signaling pathways, including Hedgehogs and Fibroblast Growth Factors. Experiments include fate mapping, time lapse analysis of neurocranial morphogenesis in the intact embryo, and genetic gain- and loss-of-function studies. Further, a new set of mutations perturbing early neurocranial patterns will be collected.

View original record on NIH RePORTER →