KIDNEY INJURY MOLECULE-1 AND DIABETIC NEPHROPATHY

$124,719U54FY2011RRNIH

Morehouse School Of Medicine, Atlanta GA

Investigators

Linked publications & trials

Paper 30968433 Paper 30166716 Paper 30099039 Paper 30059666 Paper 29940798 Paper 29593848 Paper 29259391 Paper 29247541 Paper 28941766 Paper 28805677 Paper 28497076 Paper 27596278 Paper 27314810 Paper 26912237 Paper 26780313 Paper 26332568 Paper 26305570 Paper 25880399 Paper 25849723 Paper 25383991 Paper 25203735 Paper 25192910 Paper 25046059 Paper 24910479 Paper 24903552 Paper 24772448 Paper 24223965 Paper 24106342 Paper 23996496 Paper 23974385 Paper 23795347 Paper 23758830 Paper 23617241 Paper 23589705 Paper 23560351 Paper 23504451 Paper 23402960 Paper 23313541 Paper 23246490 Paper 23234426 Paper 23224900 Paper 23201171 Paper 23181201 Paper 23063035 Paper 22900119 Paper 22900117 Paper 22820909 Paper 22643973 Paper 22643553 Paper 22488405 Paper 22262158 Paper 22248871 Paper 22212225 Paper 22162785 Paper 22156698 Paper 22115778 Paper 22102312 Paper 22102311 Paper 22013042 Paper 21929748 Paper 21840935 Paper 21822010 Paper 21707942 Paper 21519790 Paper 21482217 Paper 21439091 Paper 21386706 Paper 21366472 Paper 21344391 Paper 21305825 Paper 21045734 Paper 20964480 Paper 19900820 Paper 19880467

Abstract

This subproject is one of many research subprojects utilizing the resources provided by a Center grant funded by NIH/NCRR. Primary support for the subproject and the subproject's principal investigator may have been provided by other sources, including other NIH sources. The Total Cost listed for the subproject likely represents the estimated amount of Center infrastructure utilized by the subproject, not direct funding provided by the NCRR grant to the subproject or subproject staff. The overall goal of this project remains the same and is to elucidate the regulatory mechanism and functional role of kidney injury molecule-1 (Kim-1) in the pathogenesis of diabetic nephropathy. Experiments performed over the project period have resulted in significant progress on the two Specific Aims.

View original record on NIH RePORTER →